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Works cited by this work

The <scp>MAPK</scp> pathway across different malignancies: A new perspective

Radiation–Induced Signaling Results in Mitochondrial Impairment in Mouse Heart at 4 Weeks after Exposure to X-Rays

Chronic Inflammation in Cancer Development

Alpha-lipoic acid inhibits hepatic PAI-1 expression and fibrosis by inhibiting the TGF-β signaling pathway

p38 MAP KINASE REGULATES TNF-α PRODUCTION IN HUMAN ASTROCYTES AND MICROGLIA BY MULTIPLE MECHANISMS

X-Ray Irradiation of the Inner Ear of the Guinea Pig<i>An Electron Microscopic Study of the Degenerating Outer Hair Cells of the Organ of Corti</i>

Molecular Pathways: Adaptive Kinome Reprogramming in Response to Targeted Inhibition of the BRAF–MEK–ERK Pathway in Cancer

A Role for Mitochondrial Dysfunction in Perpetuating Radiation-Induced Genomic Instability

c-Jun N-terminal kinases (JNKs) mediate pro-inflammatory actions of microglia

NF-kappaB Signaling Pathways in Neurological Inflammation: A Mini Review

Effect of alpha-lipoic acid on radiation-induced small intestine injury in mice

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Alpha-lipoic acid effectively attenuates ionizing radiation-mediated testicular dysfunction in rats: Crosstalk of NF-ĸB, TGF-β, and PPAR-ϒ pathways

Metformin Protects Against Radiation-Induced Heart Injury and Attenuates the Upregulation of Dual Oxidase Genes Following Rat's Chest Irradiation.

NADPH Oxidase as a Target for Modulation of Radiation Response; Implications to Carcinogenesis and Radiotherapy

Functions of poly(ADP-ribose) polymerase (PARP) in DNA repair, genomic integrity and cell death

A Systematic Review of the Potential Chemoprotective Effects of Resveratrol on Doxorubicin‐Induced Cardiotoxicity: Focus on the Antioxidant, Antiapoptotic, and Anti‐Inflammatory Activities

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Activation of apoptosis signalling pathways by reactive oxygen species

Gastrointestinal Toxicities With Combined Antiangiogenic and Stereotactic Body Radiation Therapy

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Ionizing radiation-induced micronucleus formation is mediated by reactive oxygen species that are produced in a manner dependent on mitochondria, Nox1, and JNK

Superoxide dismutase 1 (SOD1) is essential for H <sub>2</sub> O <sub>2</sub> -mediated oxidation and inactivation of phosphatases in growth factor signaling