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LTRPC2 Ca2+-Permeable Channel Activated by Changes in Redox Status Confers Susceptibility to Cell Death

Yuji HaraCenter for Integrative Bioscience, The Graduate University for Advanced Studies, 444-8585, Okazaki, JapanMinoru WakamoriDepartment of Information Physiology, Okazaki 444-8585, JapanMasakazu IshiiCenter for Integrative Bioscience, Okazaki 444-8585, JapanEmi MaenoSchool of Life Science, The Graduate University for Advanced Studies, Okazaki 444-8585, JapanMotohiro NishidaCenter for Integrative Bioscience, Okazaki 444-8585, JapanTakashi YoshidaSchool of Life Science, The Graduate University for Advanced Studies, Okazaki 444-8585, JapanHisanobu YamadaCenter for Integrative Bioscience, Okazaki 444-8585, JapanShunichi ShimizuDepartment of Pathophysiology, School of Pharmaceutical Sciences, Showa University, Tokyo 142-8555, JapanEmiko MoriCenter for Integrative Bioscience, Okazaki 444-8585, JapanJun KudohDepartment of Molecular Biology, Keio University School of Medicine, Tokyo 160-8582, JapanNobuyoshi ShimizuDepartment of Molecular Biology, Keio University School of Medicine, Tokyo 160-8582, JapanHitoshi KuroseLaboratory of Pharmacology and Toxicology, Graduate School of Pharmaceutical Sciences, University of Tokyo, Tokyo 113-0033, JapanYasunobu OkadaDepartment of Cell Physiology, National Institute for Physiological Sciences, Okazaki 444-8585, JapanKeiji ImotoSchool of Life Science, The Graduate University for Advanced Studies, Okazaki 444-8585, JapanYasuo MoriCenter for Integrative Bioscience, Okazaki 444-8585, Japan
2002en
ABI

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