Reactive Oxygen Species Signaling in Response to Pathogens
Miguel Ángel Medina TorresDepartment of Biology (M.A.T.), Curriculum in Genetics (J.L.D.), Department of Microbiology and Immunology (J.L.D.), and Carolina Center for Genome Sciences (J.L.D.), University of North Carolina, Chapel Hill, North Carolina 27599–3280; and Sainsbury Laboratory, John Innes Center, Colney, Norwich NR4 7UH, United Kingdom (J.D.G.J.)Jonathan D. G. JonesDepartment of Biology (M.A.T.), Curriculum in Genetics (J.L.D.), Department of Microbiology and Immunology (J.L.D.), and Carolina Center for Genome Sciences (J.L.D.), University of North Carolina, Chapel Hill, North Carolina 27599–3280; and Sainsbury Laboratory, John Innes Center, Colney, Norwich NR4 7UH, United Kingdom (J.D.G.J.)Jeffery L. DanglDepartment of Biology (M.A.T.), Curriculum in Genetics (J.L.D.), Department of Microbiology and Immunology (J.L.D.), and Carolina Center for Genome Sciences (J.L.D.), University of North Carolina, Chapel Hill, North Carolina 27599–3280; and Sainsbury Laboratory, John Innes Center, Colney, Norwich NR4 7UH, United Kingdom (J.D.G.J.)
2006en
ABI
Abstract
The production of reactive oxygen species (ROS), via consumption of oxygen in a so-called oxidative burst, is one of the earliest cellular responses following successful pathogen recognition. Apoplastic generation of superoxide (O2−), or its dismutation product hydrogen peroxide (H2O2), has been
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