Работы, на которые ссылается эта работа

Работ: 128

Работа: Cell Death Induction and Protection by Activation of Ubiquitously Expressed Anion/Cation Channels. Part 2: Functional and Molecular Properties of ASOR/PAC Channels and Their Roles in Cell Volume Dysregulation and Acidotoxic Cell Death

Pyroptosis is driven by non-selective gasdermin-D pore and its morphology is different from MLKL channel-mediated necroptosis
Xin Chen, Wanting He, Lichen Hu +7
Статья2016Цитирований: 3
ABI
Acid- and Volume-Sensitive Chloride Currents in Microglial Cells
Michael Kittl, Katharina Helm, Marlena Beyreis +5
Статья2019Цитирований: 3
ABI
Molecular identification of a volume-regulated chloride channel
Dayue Darrel Duan, Cathy R. Winter, Suzanne Cowley +2
Статья1997Цитирований: 3
ABI
TMEM16B induces chloride currents activated by calcium in mammalian cells
Simone Pifferi, Michele Dibattista, Anna Menini
Статья2009Цитирований: 2
ABI
Antibodies against ClC7 inhibit extracellular acidification-induced Cl− currents and bone resorption activity in mouse osteoclasts
Kimiko Ohgi, Fujio Okamoto, Hiroshi Kajiya +2
Статья2010Цитирований: 2
ABI
Characteristics of ClC7 Cl− channels and their inhibition in mutant (G215R) associated with autosomal dominant osteopetrosis type II in native osteoclasts and hClcn7 gene-expressing cells
Hiroshi Kajiya, Fujio Okamoto, Kimiko Ohgi +3
Статья2009Цитирований: 2
ABI
Extracellular acidic pH-activated, outward rectifying chloride currents can be regulated by reactive oxygen species in human THP-1 monocytes
Zhijie Fu, Xuezhong Li, Qirong Wang +3
Статья2013Цитирований: 2
ABI
The ClC-3 Cl−/H+ Antiporter Becomes Uncoupled at Low Extracellular pH
James J. Matsuda, Mohammed Filali, Malia M. Collins +2
Статья2009Цитирований: 2
ABI
ANO2 is the cilial calcium-activated chloride channel that may mediate olfactory amplification
Aaron B. Stephan, Eleen Y. Shum, Sarah Hirsh +3
Статья2009Цитирований: 2
ABI
Acid-Sensitive Outwardly Rectifying Anion Channels in Human Erythrocytes
Yuliya Kucherenko, Daniel Mörsdorf, Florian Läng
Статья2009Цитирований: 2
ABI
Activation by Acidic pH of CLC-7 Expressed in Oocytes from Xenopus laevis
Linda Diewald, Johanna Rupp, Mathias Dreger +3
Статья2002Цитирований: 2
ABI
Subunit‐dependent oxidative stress sensitivity of LRRC8 volume‐regulated anion channels
Antonella Gradogna, Paola Gavazzo, Anna Boccaccio +1
Статья2017Цитирований: 2
ABI
Brain acidosis
Stig Rehncrona
Обзорная статья1985Цитирований: 2
ABI
The Effects of Extracellular Acidosis on Neurons and Glia in vitro
Steven A. Goldman, William A. Pulsinelli, Wendy Y. Clarke +2
Статья1989Цитирований: 2
ABI
Intracellular acidosis in human and experimental brain injury.
Anthony Marmarou
Статья1992Цитирований: 2
ABI
Transient receptor potential melastatin 4 and cell death
J. Marc Simard, Sang‐Ho Woo, Volodymyr Gerzanich
Обзорная статья2012Цитирований: 2
ABI
Без названия
ДругоеЦитирований: 2
ABI
Regulation of Human CLC-3 Channels by Multifunctional Ca2+/Calmodulin-dependent Protein Kinase
Ping Huang, Jie Liu, Anke Di +4
Статья2001Цитирований: 2
ABI
ClC‐3B, a novel ClC‐3 splicing variant that interacts with EBP50 and facilitates expression of CFTR‐regulated ORCC
Takehiko Ogura, Tetsushi Furukawa, Tetsuya Toyozaki +5
Статья2002Цитирований: 2
ABI
Acidic extracellular microenvironment and cancer
Yukio Kato, Shigeyuki Ozawa, Chihiro Miyamoto +4
Статья2013Цитирований: 2
ABI
LTRPC7 is a Mg·ATP-regulated divalent cation channel required for cell viability
Monica J. S. Nadler, Meredith C. Hermosura, Kazunori Inabe +8
Статья2001Цитирований: 2
ABI
A proton-gated cation channel involved in acid-sensing
Rainer Waldmann, Guy Champigny, Frédéric Bassilana +2
Статья1997Цитирований: 2
ABI
TRPM7 Provides an Ion Channel Mechanism for Cellular Entry of Trace Metal Ions
Mahealani K. Monteilh‐Zoller, Meredith C. Hermosura, Monica J. S. Nadler +3
Статья2002Цитирований: 2
ABI
H<sup>+</sup>‐Gated Cation Channels<sup>a</sup>
Rainer Waldmann, Guy Champigny, Éric Lingueglia +3
Обзорная статья1999Цитирований: 2
ABI
CLC Chloride Channels and Transporters: Structure, Function, Physiology, and Disease
Thomas J. Jentsch, Michael Pusch
Обзорная статья2018Цитирований: 2
ABI