The cardiac ryanodine receptor, but not sarcoplasmic reticulum Ca2+-ATPase, is a major determinant of Ca2+ alternans in intact mouse hearts
Bo SunFrom the Libin Cardiovascular Institute of Alberta, Department of Physiology and Pharmacology, University of Calgary, Calgary, Alberta T2N 4N1, CanadaJinhong WeiFrom the Libin Cardiovascular Institute of Alberta, Department of Physiology and Pharmacology, University of Calgary, Calgary, Alberta T2N 4N1, CanadaXiaowei ZhongFrom the Libin Cardiovascular Institute of Alberta, Department of Physiology and Pharmacology, University of Calgary, Calgary, Alberta T2N 4N1, CanadaWenting GuoFrom the Libin Cardiovascular Institute of Alberta, Department of Physiology and Pharmacology, University of Calgary, Calgary, Alberta T2N 4N1, CanadaJinjing YaoFrom the Libin Cardiovascular Institute of Alberta, Department of Physiology and Pharmacology, University of Calgary, Calgary, Alberta T2N 4N1, CanadaRuiwu WangFrom the Libin Cardiovascular Institute of Alberta, Department of Physiology and Pharmacology, University of Calgary, Calgary, Alberta T2N 4N1, CanadaAlexander Vallmitjanathe Department of Automatic Control, Universitat Politècnica de Catalunya, Barcelona 08034, Spain, andRaúl Benítezthe Department of Automatic Control, Universitat Politècnica de Catalunya, Barcelona 08034, Spain, andLeif Hove‐Madsenthe Biomedical Research Institute of Barcelona (IIBB), CSIC, Sant Pau, Hospital de Sant Pau, Barcelona 08025, SpainS.R. Wayne ChenFrom the Libin Cardiovascular Institute of Alberta, Department of Physiology and Pharmacology, University of Calgary, Calgary, Alberta T2N 4N1, Canada, [email protected]
2018en
ABI
Аннотация
alternans in intact working hearts, making RyR2 an important therapeutic target for cardiac alternans.
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