WAYS TO OPTIMIZE REHABILITATION ACTIVITIES IN HEMORRHAGIC AND ISCHEMIC STROKE

The development of acute cerebral ischemia triggers pathobiochemical cascade reactions, the outcome of which is cerebral infarction (MI), which is formed by two mechanisms: necrotic cell death and apoptosis - programmed cell death. These modern pathogenetic concepts have made it possible to propose a sequence of stages »Based on their causal relationships. Each stage of the cascade is a target for the therapeutic effect of drugs, primarily with neuroprotective effects.

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