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Иш: Dual role of miR-155 and exosomal miR-155 in tumor angiogenesis: implications for cancer progression and therapy
The Role of Exosomes in Breast Cancer
Michelle C. Lowry, William M. Gallagher, Lorraine O’Driscoll
Шарҳ мақола20154 иқтибосABIExosome and Exosomal MicroRNA: Trafficking, Sorting, and Function
Jian Zhang, Sha Li, Lu Li +4
Шарҳ мақола20154 иқтибосABImicroRNA Regulation of Inflammatory Responses
Ryan M. O’Connell, Dinesh S. Rao, David Baltimore
Шарҳ мақола20123 иқтибосABIExosomes in cancer development and clinical applications
Yuling Tai, Ko‐Chien Chen, Jer‐Tsong Hsieh +1
Шарҳ мақола20183 иқтибосABIExosomes: Extracellular organelles important in intercellular communication
Suresh Mathivanan, Hong Ji, Richard J. Simpson
Шарҳ мақола20102 иқтибосABIMicroRNA-155 Is Involved in the Pathogenesis of Ulcerative Colitis by Targeting FOXO3a
Min Min, Lihua Peng, Yunsheng Yang +3
Мақола20142 иқтибосABIMicroRNA-503 targets FGF2 and VEGFA and inhibits tumor angiogenesis and growth
Bisheng Zhou, Ruihua Ma, Wenxia Si +4
Мақола20132 иқтибосABICancer-Associated Fibroblasts: Their Characteristics and Their Roles in Tumor Growth
Kazuyoshi Shiga, Masayasu Hara, Takaya Nagasaki +3
Шарҳ мақола20152 иқтибосABITumor angiogenesis and vascular normalization: alternative therapeutic targets
Claire Viallard, Bruno Larrivée
Шарҳ мақола20172 иқтибосABIAn NF-κB-microRNA regulatory network tunes macrophage inflammatory responses
Mati Mann, Arnav Mehta, Jimmy L. Zhao +6
Мақола20172 иқтибосABIMicroRNA-155—at the Critical Interface of Innate and Adaptive Immunity in Arthritis
Stefano Alivernini, Elisa Gremese, Charles McSharry +4
Шарҳ мақола20182 иқтибосABIMiR-155-5p inhibits PDK1 and promotes autophagy via the mTOR pathway in cervical cancer
Fang Wang, Shu Shan, Yan Huo +6
Мақола20182 иқтибосABImiRNAs reshape immunity and inflammatory responses in bacterial infection
Xikun Zhou, Xuefeng Li, Min Wu
Мақола20182 иқтибосABIMicroRNA-mediated regulation of glucose and lipid metabolism
Pamela Agbu, Richard W. Carthew
Шарҳ мақола20212 иқтибосABI