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7 та иш

Иш: Chloride channels in myocytes from rabbit colon are regulated by a pertussis toxin-sensitive G protein

  1. The maxi-anion channel: a classical channel playing novel roles through an unidentified molecular entity

    Ravshan Z. Sabirov, Yasunobu Okada

    Шарҳ мақолаIon channel regulation and functionThe Journal of Physiological Sciences200915 иқтибос
    ABI

  2. The properties, functions, and pathophysiology of maxi-anion channels

    Ravshan Z. Sabirov, Petr G. Merzlyak, Md. Rafiqul Islam +2

    Шарҳ мақолаIon channel regulation and functionPflügers Archiv - European Journal of Physiology201614 иқтибос
    ABI

  3. Cell Volume-Activated and Volume-Correlated Anion Channels in Mammalian Cells: Their Biophysical, Molecular, and Pharmacological Properties

    Yasunobu Okada, Toshiaki Okada, Kaori Sato +8

    Шарҳ мақолаIon channel regulation and functionPharmacological Reviews201812 иқтибос
    ABI

  4. Activation of maxi-anion channel by protein tyrosine dephosphorylation

    Abduqodir Toychiev, Ravshan Z. Sabirov, Nobuyuki Takahashi +5

    МақолаProtein Tyrosine PhosphatasesAmerican Journal of Physiology-Cell Physiology200910 иқтибос
    ABI

  5. Molecular Identities and ATP Release Activities of Two Types of Volume-Regulatory Anion Channels, VSOR and Maxi-Cl

    Yasunobu Okada, Toshiaki Okada, Md. Rafiqul Islam +1

    Шарҳ мақолаIon channel regulation and functionCurrent topics in membranes201810 иқтибос
    ABI

  6. Roles of volume-regulatory anion channels, VSOR and Maxi-Cl, in apoptosis, cisplatin resistance, necrosis, ischemic cell death, stroke and myocardial infarction

    Yasunobu Okada, Tomohiro Numata, Kaori Sato +4

    Шарҳ мақолаIon channel regulation and functionCurrent topics in membranes20198 иқтибос
    ABI

  7. The ATP-Releasing Maxi-Cl Channel: Its Identity, Molecular Partners, and Physiological/Pathophysiological Implications

    Ravshan Z. Sabirov, Md. Shahidul Islam, Toshiaki Okada +4

    Шарҳ мақолаIon channel regulation and functionLife20215 иқтибос
    ABI