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Ish: Polyglutamine-induced ion channels: a possible mechanism for the neurotoxicity of huntington and other CAG repeat diseases
Channel Formation by a Neurotoxic Prion Protein Fragment
Meng‐Chin Lin, Tajib A. Mirzabekov, Bruce L. Kagan
MaqolaOchiq kirish19973 iqtibosABIPolyglutamines, nuclear inclusions and neurodegeneration
Astrid Lunkes, Jean‐Louis Mandel
Maqola19972 iqtibosABIGlutamine repeats as polar zippers: their possible role in inherited neurodegenerative diseases.
M. F. Perutz, T. Johnson, Masashi Suzuki +1
MaqolaOchiq kirish19942 iqtibosABISynthetic mammalian C‐type natriuretic peptide forms large cation channels
MaqolaOchiq kirish19992 iqtibosABIHuntingtin-Encoded Polyglutamine Expansions Form Amyloid-like Protein Aggregates In Vitro and In Vivo
Eberhard Scherzinger, Rudi Lurz, Mark Turmaine +7
MaqolaOchiq kirish19972 iqtibosABIPore Formation by the Cytotoxic Islet Amyloid Peptide Amylin
Tajib A. Mirzabekov, Meng‐Chin Lin, Bruce L. Kagan
MaqolaOchiq kirish19962 iqtibosABI