LTRPC7 is a Mg·ATP-regulated divalent cation channel required for cell viability
Monica J. S. NadlerDepartment of Pathology, Beth Israel Deaconess Medical Center and Harvard Medical School, Boston, Massachusetts 02215, USAMeredith C. HermosuraLaboratory of Cell and Molecular Signaling, Center for Biomedical Research at The Queen's Medical Center and John A. Burns School of Medicine at the University of Hawaii, Honolulu, USAKazunori InabeDepartment of Molecular Genetics, Institute for Liver Research, Kansai Medical University, Osaka, 570-8506, JapanAnne‐Laure PerraudDepartment of Pathology, Beth Israel Deaconess Medical center and Harvard Medical School, Boston, USAQiqin ZhuDepartment of Pathology, Beth Israel Deaconess Medical Center and Harvard Medical School, Boston, 02215, Massachusetts, USAAlexander J. StokesDepartment of Pathology, Beth Israel Deaconess Medical Center and Harvard Medical School, Boston, 02215, Massachusetts, USATomohiro KurosakiDepartment of Molecular Genetics, Institute for Liver Research, Kansai Medical University, Osaka, 570-8506, JapanJean-Pierre KinetDepartment of Pathology, Beth Israel Deaconess Medical Center and Harvard Medical School, Boston, 02215, Massachusetts, USAReinhold PennerLaboratory of Cell and Molecular Signaling, Center for Biomedical Research at The Queen's Medical Center and John A. Burns School of Medicine at the University of Hawaii, Honolulu, 96813, Hawaii, USAAndrew M. ScharenbergDepartment of Pathology, Beth Israel Deaconess Medical Center and Harvard Medical School, Boston, USAAndrea FleigLaboratory of Cell and Molecular Signaling, Center for Biomedical Research at The Queen's Medical Center and John A. Burns School of Medicine at the University of Hawaii, Honolulu, 96813, Hawaii, USA
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