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Работы, на которые ссылается эта работа

Работ: 110

Работа: miR-155 and the orchestration of cell-death evasion and chemoresistance in cancer: Isoform-specific mechanisms and therapeutic opportunities

  1. Hallmarks of Cancer: The Next Generation

    Douglas Hanahan, Robert A. Weinberg

    Обзорная статья2011Цитирований: 18
    ABI
  2. Virus, Exosome, and MicroRNA: New Insights into Autophagy

    Javid Sadri Nahand, Arash Salmaninejad, Samaneh Mollazadeh +15

    Статья2022Цитирований: 18
    ABI
  3. Cell death pathways and viruses: Role of microRNAs

    Javid Sadri Nahand, Layla Shojaie, Seyed Amirreza Akhlagh +9

    Обзорная статья2021Цитирований: 14
    ABI
  4. MiRNA-related metastasis in oral cancer: moving and shaking

    Meghdad Eslami, Saba Khazeni, Xaniar Mohammadi Khanaghah +9

    Обзорная статья2023Цитирований: 12
    ABI
  5. The tumor microenvironment's gambit: Exosomal pawns on the board of head and neck cancer

    Solmaz Mohamadi, Parisa Mehrasa, Bahareh Mehramuz +5

    Обзорная статья2024Цитирований: 12
    ABI
  6. Exosomal miRNAs: the tumor's trojan horse in selective metastasis

    Mobina Bayat, Javid Sadri Nahand

    Обзорная статья2024Цитирований: 11
    ABI
  7. Regulatory role of microRNAs in virus-mediated inflammation

    Hossein Bannazadeh Baghi, Mobina Bayat, Parisa Mehrasa +9

    Обзорная статья2024Цитирований: 8
    ABI
  8. Oncogenic viruses and chemoresistance: What do we know?

    Javid Sadri Nahand, Nikta Rabiei, Reza Fathazam +10

    Обзорная статья2021Цитирований: 7
    ABI
  9. MicroRNA therapeutics: towards a new era for the management of cancer and other diseases

    Rajesha Rupaimoole, Frank J. Slack

    Обзорная статья2017Цитирований: 6
    ABI
  10. MicroRNA-21 Regulates Expression of the PTEN Tumor Suppressor Gene in Human Hepatocellular Cancer

    Fanyin Meng, Roger Henson, Hania Wehbe–Janek +3

    Статья2007Цитирований: 6
    ABI
  11. Let’s make it personal: CRISPR tools in manipulating cell death pathways for cancer treatment

    Mobina Bayat, Javid Sadri Nahand

    Обзорная статья2024Цитирований: 6
    ABI
  12. Targeting apoptosis in cancer therapy

    Benedito A. Carneiro, Wafik S. El‐Deiry

    Обзорная статья2020Цитирований: 5
    ABI
  13. Battlegrounds of treatment resistance: decoding the tumor microenvironment

    Mobina Bayat, Javid Sadri Nahand

    Обзорная статья2025Цитирований: 5
    ABI
  14. Tumor Suppressor miRNA in Cancer Cells and the Tumor Microenvironment: Mechanism of Deregulation and Clinical Implications

    Khalid Otmani, Philippe Lewalle

    Обзорная статья2021Цитирований: 5
    ABI
  15. Exosomal regulation of cellular reprogramming and polarization in the tumor microenvironment

    Mobina Bayat, Parviz Shahabi, Tohid Rezaei +1

    Статья2025Цитирований: 5
    ABI
  16. To be or not to be: navigating the influence of MicroRNAs on cervical cancer cell death

    Mohammad Taghizadieh, Masoumeh Kalantari, Roksana Bakhshali +6

    Обзорная статья2025Цитирований: 4
    ABI
  17. MicroRNA-155—at the Critical Interface of Innate and Adaptive Immunity in Arthritis

    Stefano Alivernini, Elisa Gremese, Charles McSharry +4

    Обзорная статья2018Цитирований: 2
    ABI
  18. MiR-155-5p inhibits PDK1 and promotes autophagy via the mTOR pathway in cervical cancer

    Fang Wang, Shu Shan, Yan Huo +6

    Статья2018Цитирований: 2
    ABI
  19. miR-155 Accelerates the Growth of Human Liver Cancer Cells by Activating CDK2 via Targeting H3F3A

    Xiaoru Xin, Yanan Lu, Sijie Xie +11

    Статья2020Цитирований: 2
    ABI
  20. <i>miR-15</i> and <i>miR-16</i> induce apoptosis by targeting BCL2

    Amelia Cimmino, George A. Calin, Muller Fabbri +14

    Статья2005Цитирований: 2
    ABI
  21. miR-155-5p increases the sensitivity of liver cancer cells to adriamycin by regulating ATG5-mediated autophagy

    Yu Qiu, Xiaoping Xu, Xinmin Yin +1

    Статья2020Цитирований: 2
    ABI
  22. Necroptosis: Mechanisms and Relevance to Disease

    Lorenzo Galluzzi, Oliver Kepp, Francis Ka-Ming Chan +1

    Обзорная статья2016Цитирований: 2
    ABI